Archive for the 'Environmental Asbestos Exposure' Category

June 4th, 2008. Mesothelioma due to environmental exposure to erionite in Turkey
It has also been confirmed that a genetic predisposition to erionite carcinogenesis is the cause of the mesothelioma epidemic in Cappadocia. Summary: The data obtained recently on the epidemiology, etiology, and pathogenesis of the mesothelioma due to erionite exposure in Turkey are described.
May 30th, 2008. Two cases of asbestosis and one case of rounded atelectasis due to non-occupational asbestos exposure
The three cases indicate that non-neoplastic, long-latency asbestos-related diseases which are usually observed as a consequence of occupational exposures, may rarely develop in subjects living in contaminated geographical sites and buildings. These unusual environmental diseases raise the diagnostic problem of differentiating them from other, more common respiratory illnesses, and impose the duties of patient notification, assessment and follow-up, to assess the possibility of progression of disease and increased neoplastic risk.
May 29th, 2008. Erionite-induced pleural plaques : Exposition to urban pollution in a female Turkish migrant in Germany
Short-term clinical and radiologic control of the patient will be necessary because of the risk of MPM. For epidemiologic considerations discussed referring to current literature, a growing incidence of this type of disease in migrants from high-risk areas must be reckoned with in Germany, even without exposition to asbestos.
April 16th, 2008. Exposition to asbestos dust in agricultural environment. Some significant exposure forms documented by the Hospital Unit of Occupational Medicine of Hospital Institute of Cremona
At the moment there aren't full-blown asbestos-linked pathology but these workers are potentially at risk of getting them. So it has an importance both on individual and global level for the rural area near Cremona.
April 16th, 2008. Case studies on the malignant mesothelioma in Pellezzano (SA
All five patients answered the questionnaire and a team of doctors performed an on-the-spot investigation. The studies verified the existence of two kind of asbestos exposure, professional (Cotton Manufacture, Construction, Foundry) and environmental (cement-asbestos pre-fabricated since 1980 earthquake), that have to be analyzed further.
April 9th, 2008. An evaluation of the risks of lung cancer and mesothelioma from exposure to amphibole cleavage fragments
Lawrence, NY talc does not produce mesotheliomas in animals while amphibole asbestos does. In sum, the weight of evidence fully supports a conclusion that non-asbestiform amphiboles do not increase the risk of lung cancer or mesothelioma.
March 19th, 2008. Erionite series minerals: mineralogical and carcinogenic properties
The larger surface area creates a wider platform for mineral-cell interaction and thus more possibilities of proliferative transformation of mesothelial cells. Consequently, understanding the exact mineralogical properties will help determination of the true carcinogenic mechanism(s) of the mineral for prevention and possibly treatment of malignant mesothelioma.
January 26th, 2008. Translocation pathways for inhaled asbestos fibers
25 microns) can travel larger distances due to low steric hindrance (in mesothelioma about 90% of fibers are ultrafine). Fibers translocation is a slow process developing over decades of life: it is aided by high biopersistence, by inflammation-induced increase in permeability, by low steric hindrance and by fibers motion pattern at low Reynolds numbers; it is hindered by fibrosis that increases interstitial flow resistances.
January 22nd, 2008. Risk assessment due to environmental exposures to fibrous particulates associated with taconite ore
77 excess cancer case in 1,000,000 lifetimes. Finally, we briefly review the epidemiology studies of grunerite asbestos (amosite) focusing on the exposure conditions associated with increased risk of human mesothelioma.
January 17th, 2008. Epidemiology of pleural mesothelioma in a population with non-occupational asbestos exposure
Conclusions: Environmental exposure to asbestos begins at birth and this may be important in the age of disease onset, if a threshold model for cancer initiation is operative. Both men and women had an excess risk of mesothelioma. Given that a family history of MPM was not common in this relatively homogenous patient group, a genetic predisposition to mesothelioma appears unlikely.
January 4th, 2008. Environmental mesothelioma associated with tremolite asbestos: Lessons from the experiences of Turkey, Greece, Corsica, New Caledonia and Cyprus
It can also elucidate the effects of fibers that behave like amphibole asbestos. However, this type of exposure is not useful for studying the potential effects of small amounts of asbestos in the ambient air of big cities due to the differing nature of the fiber types and modes of exposure between the regions.
December 22nd, 2007. Cancer incidence among women and girls environmentally and occupationally exposed to blue asbestos at Wittenoom, Western Australia
A significant exposure-response relationship was present for mesothelioma, but not for lung cancer. Forty years after the asbestos mine and mill at Wittenoom were closed, there is a high toll from cancer among the former female residents of the town and company workers.
December 20th, 2007. Asbestos related diseases in Italy: an integrated approach to identify unexpected professional or environmental exposure risks at municipal level
Conclusions: Availability long-term national figures and the different etiology of asbestos related diseases are the key features of this exercise that was applied to Italy, but can be replicated wherever registration systems of diseases related to long term exposure to asbestos are available.
December 12th, 2007. Mesothelioma incidence around Goor, The Netherlands, and far beyond, the asbestos drama unfolds
The WHO and the International Labour Office are collaborating towards the elimination of asbestos-related diseases worldwide. More awareness is needed in order to prevent similar tragedies occurring from other side effects of work.
December 12th, 2007. Environmental exposure to asbestos in the area around Goor has been established as the cause of pleural mesothelioma in women
Conclusion: The environmental pollution to asbestos waste materials in the area around Goor was the main cause of the strongly increased incidence of pleural mesothelioma among women in this area. Taking into account an equal risk among men, the consequences of asbestos exposure in the area around Goor in the next 25 years are likely to result in 2 cases of pleural mesothelioma each year.
November 21st, 2007. South African experience with asbestos related environmental mesothelioma: Is asbestos fiber type important?
Crocidolite appears to be far more mesotheliomagenic than amosite, and chrysotile has not been implicated in the disease. This is true for both occupationally and environmentally exposed individuals.
November 6th, 2007. Analysis of latency time and its determinants in asbestos related malignant mesothelioma cases of the Italian register
6 years increasing in recent years with a linear trend. Anatomical site, gender and morphology were not relevant for MM latency time whereas a shorter latency period was documented among occupationally exposed subjects (43 years) with respect to environmentally and household exposed ones (48 years).
October 24th, 2007. Peritoneal mesothelioma: a review
The incidence has increased in the past 2 decades. Only 20% to 33% of all mesotheliomas arise from the peritoneum itself; the pleura is the most common site of origin.
October 2nd, 2007. Genetic susceptibility to malignant mesothelioma and exposure to asbestos: The influence of the familial factor
Conclusions: The evaluation of the published materials supports the hypothesis that – although familial clustering of MM is largely attributable to shared asbestos exposure – the additional contribution of factors dealing with genetic susceptibility may play a role in the etiology of MM.
September 19th, 2007. SV40 Multiple Tissue Infection and Asbestos Exposure in a Hyperendemic Area for Malignant Mesothelioma
045). This survey shows that SV40 sustains infections in multiple tissues in malignant mesothelioma patients from a geographic area affected with asbestos-related mesothelioma.
|
|  |